Validating Stat3 in cancer therapy
- 1 June 2005
- journal article
- editorial
- Published by Springer Nature in Nature Medicine
- Vol. 11 (6) , 595-596
- https://doi.org/10.1038/nm0605-595
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Stat3 is required for ALK-mediated lymphomagenesis and provides a possible therapeutic targetNature Medicine, 2005
- A low-molecular-weight compound discovered through virtual database screening inhibits Stat3 function in breast cancer cellsProceedings of the National Academy of Sciences, 2005
- Anaphase-promoting complex-dependent proteolysis of cell cycle regulators and genomic instability of cancer cellsOncogene, 2005
- In vivo antitumor efficacy of STAT3 blockade using a transcription factor decoy approach: implications for cancer therapyOncogene, 2004
- The STATs of cancer — new molecular targets come of ageNature Reviews Cancer, 2004
- SOCS-3 is frequently silenced by hypermethylation and suppresses cell growth in human lung cancerProceedings of the National Academy of Sciences, 2003
- Transcription factors as targets for cancer therapyNature Reviews Cancer, 2002
- STATs: transcriptional control and biological impactNature Reviews Molecular Cell Biology, 2002
- Stat3 as an OncogenePublished by Elsevier ,1999
- Fusion of a Kinase Gene, ALK , to a Nucleolar Protein Gene, NPM , in Non-Hodgkin's LymphomaScience, 1994