Vitamin D Receptor Expression Is Linked to Cell Cycle Control in Normal Human Keratinocytes
- 1 December 2000
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 279 (1) , 89-94
- https://doi.org/10.1006/bbrc.2000.3892
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Suppression of Vitamin D Receptor and Induction of Retinoid X Receptor α Expression During Squamous Differentiation of Cultured KeratinocytesJournal of Investigative Dermatology, 2000
- The Flavonoid Apigenin Suppresses Vitamin D Receptor Expression and Vitamin D Responsiveness in Normal Human KeratinocytesBiochemical and Biophysical Research Communications, 2000
- Normalization of Mineral Ion Homeostasis by Dietary Means Prevents Hyperparathyroidism, Rickets, and Osteomalacia, But Not Alopecia in Vitamin D Receptor-Ablated Mice1Endocrinology, 1998
- Anchorage-Dependent Expression of the Vitamin D Receptor in Normal Human KeratinocytesJournal of Investigative Dermatology, 1998
- Ultraviolet B Suppresses Vitamin D Receptor Gene Expression in KeratinocytesBiochemical and Biophysical Research Communications, 1998
- Mice lacking the vitamin D receptor exhibit impaired bone formation, uterine hypoplasia and growth retardation after weaningNature Genetics, 1997
- Retinoic Acid Modulates the Anti-Proliferative Effect of 1,25-Dihydroxyvitamin D3 in Cultured Human Epidermal KeratinocytesJournal of Investigative Dermatology, 1997
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- Human D-type cyclinCell, 1991
- Monoclonal antibodies to the porcine intestinal receptor for 1,25-dihydroxyvitamin D3: interaction with distinct receptor domainsBiochemistry, 1986