Src Kinases Mediate STAT Growth Pathways in Squamous Cell Carcinoma of the Head and Neck
Open Access
- 1 August 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (34) , 31574-31583
- https://doi.org/10.1074/jbc.m303499200
Abstract
No abstract availableKeywords
This publication has 57 references indexed in Scilit:
- Overexpression of c-Src in areas of hyperproliferation in head and neck cancer, premalignant lesions and benign mucosal disordersJournal of Oral Pathology & Medicine, 2007
- Activation of STAT3 by the Src Family Kinase Hck Requires a Functional SH3 DomainJournal of Biological Chemistry, 2002
- Phosphorylation of STAT-3 in Response to Basic Fibroblast Growth Factor Occurs through a Mechanism Involving Platelet-activating Factor, JAK-2, and Src in Human Umbilical Vein Endothelial CellsPublished by Elsevier ,2002
- Differential function of STAT5 isoforms in head and neck cancer growth controlOncogene, 2002
- Activation of Extracellular Signal-regulated Kinase by Ultraviolet Is Mediated through Src-dependent Epidermal Growth Factor Receptor PhosphorylationJournal of Biological Chemistry, 2002
- STATs in oncogenesisOncogene, 2000
- STAT 1α/1β, STAT 3 and STAT 5: Expression and Association with c-MET and EGF-Receptor in Long-Term Cultures of Human HepatocytesBiochemical and Biophysical Research Communications, 1999
- Inhibition of acute lymphoblastic leukaemia by a Jak-2 inhibitorNature, 1996
- TRANSCRIPTIONAL RESPONSES TO POLYPEPTIDE LIGANDS: The JAK-STAT PathwayAnnual Review of Biochemistry, 1995
- EGF Receptor Deletions Define a Region Specifically Mediating Stat Transcription Factor ActivationBiochemical and Biophysical Research Communications, 1995