Mechanisms Leading to Sustained Reversion of β-Thalassemia in Mice by Doxycycline-Controlled Epo Delivery from Muscles
Open Access
- 31 December 2002
- journal article
- Published by Elsevier in Molecular Therapy
- Vol. 6 (6) , 793-803
- https://doi.org/10.1006/mthe.2002.0810
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Ineffective erythropoiesis in β-thalassemia major is due to apoptosis at the polychromatophilic normoblast stageExperimental Hematology, 2000
- Hemoglobin switching in unicellular erythroid culture of sibling erythroid burst-forming units: kit ligand induces a dose-dependent fetal hemoglobin reactivation potentiated by sodium butyrateBlood, 2000
- Treatment of thalassaemia major with phenylbutyrate and hydroxyureaThe Lancet, 1997
- Abnormal assembly of membrane proteins in erythroid progenitors of patients with beta-thalassemia majorBlood, 1996
- Thrombopoietin expands erythroid progenitors, increases red cell production, and enhances erythroid recovery after myelosuppressive therapy.Journal of Clinical Investigation, 1995
- Serum erythropoietin and erythropoiesis in high- and low-fetal hemoglobin beta-thalassemia intermedia patientsBlood, 1994
- Effects of interleukin-3 and erythropoietin on in vivo erythropoiesis and F-cell formation in primatesBlood, 1989
- Erythropoietin activity in the serum of beta thalassemic patientsScandinavian Journal of Haematology, 1986
- Fetal Hb Production during Acute Erythroid Expansion: I. OBSERVATIONS IN PATIENTS WITH TRANSIENT ERYTHROBLASTOPENIA AND POST‐PHLEBOTOMYBritish Journal of Haematology, 1980
- Fetal erythropoiesis following bone marrow transplantationBlood, 1976