Abstract
Under ad-lib self-selection conditions desoxycorticosterone-treated rats ingested larger amounts of . 25 M saline but smaller amounts of 1.00M saline than did untreated adrenalectomized rats. When saline intake of desoxycorticosterone-treated and sodium-deficient (sodium-deprived adrenalectomized or formalin-treated) rats was compared using a brief access procedure in order to hold drive level more constant, there were no significant differences between the groups in amount of saline ingested as a function of the concentration of the solution. It was concluded that a previously proposed hypothesis that a single mechanism underlies both desoxycorticosterone- and deficiency-induced sodium appetite remains tenable.

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