Genetic basis of the effects of ultraviolet light B on cutaneous immunity. Evidence that polymorphism at the Tnfa and Lps loci governs susceptibility
- 1 December 1990
- journal article
- Published by Springer Nature in Immunogenetics
- Vol. 32 (6) , 398-405
- https://doi.org/10.1007/bf00241633
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Analysis of restriction fragment length polymorphism in lymphokine genes of normal and autoimmune mice.The Journal of Experimental Medicine, 1989
- Promotion and Subsequent Inhibition of Plasminogen Activation after Administration of Intravenous Endotoxin to Normal SubjectsNew England Journal of Medicine, 1989
- Detection of Circulating Tumor Necrosis Factor after Endotoxin AdministrationNew England Journal of Medicine, 1988
- Tumour necrosis factor-α in murine autoimmune 'lupus' nephritisNature, 1988
- Granulocyte/macrophage colony-stimulating factor is essential for the viability and function of cultured murine epidermal Langerhans cells.The Journal of Experimental Medicine, 1987
- Tumor Necrosis Factor: A Cytokine Involved in Toxic Effects of EndotoxinClinical Infectious Diseases, 1987
- Antigen-Presenting OKM5+ Melanophages Appear in Human Epidermis After Ultraviolet RadiationJournal of Investigative Dermatology, 1986
- Enhancement of the Elicitation Phase of the Murine Contact Hypersensitivity Response by Prior Exposure to Local Ultraviolet RadiationJournal of Investigative Dermatology, 1986
- Malignant melanoma in southern Arizona. Increasing incidence and sunlight as an etiologic factorArchives of Dermatology, 1981
- Genetic Control of Leucocyte Responses to EndotoxinNature, 1968