Transient cerebral ischemia decreases calcium/ calmodulin-dependent protein kinase II immunoreactivity, but not mRNA levels in the gerbil hippocampus
- 24 December 1995
- journal article
- Published by Elsevier in Brain Research
- Vol. 705 (1-2) , 307-314
- https://doi.org/10.1016/0006-8993(95)01155-2
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Locomotor activity in the ischemic gerbilBrain Research, 1993
- Regulation of type-II calmodulin kinase: Functional implicationsBrain Research Reviews, 1993
- Neurotensin-induced hypothermia prevents hippocampal neuronal damage and increased locomotor activity in ischemic gerbilsBrain Research Bulletin, 1993
- Ischemia‐Induced Translocation of Ca2+/Calmodulin‐Dependent Protein Kinase II: Potential Role in Neuronal DamageJournal of Neurochemistry, 1992
- A brief period of hypoxia causes proteolysis of cytoskeletal proteins in hippocampal slicesBrain Research, 1991
- In situ hybridization histochemistry of Ca2+/calmodulin-dependent protein kinase in developing rat brainJournal of Neuroscience, 1990
- The importance of brain temperature in cerebral ischemic injury.Stroke, 1989
- Postischemic moderate hypothermia inhibits CA1 hippocampal ischemic neuronal injuryNeuroscience Letters, 1989
- A Retinal Calmodulin‐Dependent Kinase: Calcium/ Calmodulin‐Stimulated and ‐Inhibited StatesJournal of Neurochemistry, 1988
- Small Differences in Intraischemic Brain Temperature Critically Determine the Extent of Ischemic Neuronal InjuryJournal of Cerebral Blood Flow & Metabolism, 1987