Effect of p53 Gene Therapy Combined with CTLA4Ig Selective Immunosuppression on Prolonged Neointima Formation Reduction in a Rat Model
- 1 July 2000
- journal article
- research article
- Published by Elsevier in Annals of Vascular Surgery
- Vol. 14 (4) , 385-392
- https://doi.org/10.1007/s100169910077
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- P53 gene transfer to the injured rat carotid artery promotes apoptosisSurgery, 1999
- In vivo gene transfer into rat arterial walls with novel adeno-associated virus vectorsJournal of Vascular Surgery, 1997
- Antisense basic fibroblast growth factor gene transfer reduces neointimal thickening after arterial injuryJournal of Vascular Surgery, 1997
- Established immunity precludes adenovirus-mediated gene transfer in rat carotid arteries. Potential for immunosuppression and vector engineering to overcome barriers of immunity.Journal of Clinical Investigation, 1997
- Adenovirus-mediated over-expression of the cyclin/cyclin-dependent kinase inhibitor, p21 inhibits vascular smooth muscle cell proliferation and neointima formation in the rat carotid artery model of balloon angioplasty.Journal of Clinical Investigation, 1995
- Consequences of p53 Gene Expression by Adenovirus Vector on Cell Cycle Arrest and Apoptosis in Human Aortic Vascular Smooth Muscle CellsBiochemical and Biophysical Research Communications, 1995
- Long–term hepatic adenovirus–mediated gene expression in mice following CTLA4Ig administrationNature Genetics, 1995
- CTLA4Ig TREATMENT AMELIORATES THE LETHALITY OF MURINE GRAFT-VERSUS-HOST DISEASE ACROSS MAJOR HISTOCOMPATIBILITY COMPLEX BARRIERSTransplantation, 1994
- Gene Therapy for Vascular Smooth Muscle Cell Proliferation After Arterial InjuryScience, 1994
- Long-Term Survival of Xenogeneic Pancreatic Islet Grafts Induced by CTLA4lgScience, 1992