HIV Tat Activates c-Jun Amino-terminal Kinase through an Oxidant-Dependent Mechanism
- 1 July 2001
- Vol. 286 (1) , 62-71
- https://doi.org/10.1006/viro.2001.0998
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Clinical course of cardiomyopathy in HIV-infected patients with or without encephalopathy related to the myocardial expression of tumour necrosis factor-α and nitric oxide synthaseAIDS, 2000
- Mechanotransduction in Response to Shear StressJournal of Biological Chemistry, 1999
- Human Immunodeficiency Virus Tat Modulates the Flk-1/KDR Receptor, Mitogen-Activated Protein Kinases, and Components of Focal Adhesion in Kaposi’s Sarcoma CellsJournal of Virology, 1998
- G-protein-coupled receptor of Kaposi's sarcoma-associated herpesvirus is a viral oncogene and angiogenesis activatorNature, 1998
- Redox-regulated Signaling by Lactosylceramide in the Proliferation of Human Aortic Smooth Muscle CellsJournal of Biological Chemistry, 1997
- Arachidonic acid activates c-jun N-terminal kinase through NADPH oxidase in rabbit proximal tubular epithelial cellsProceedings of the National Academy of Sciences, 1997
- The angiogenesis induced by HIV–1 Tat protein is mediated by the Flk–1/KDR receptor on vascular endothelial cellsNature Medicine, 1996
- Angiogenic properties of human immunodeficiency virus type 1 Tat protein.Proceedings of the National Academy of Sciences, 1995
- Tat protein of human immunodeficiency virus type 1 represses expression of manganese superoxide dismutase in HeLa cells.Proceedings of the National Academy of Sciences, 1993
- AIDS-Kaposi's Sarcoma-Derived Cells Express Cytokines with Autocrine and Paracrine Growth EffectsScience, 1989