Bcl-2 Undergoes Phosphorylation by c-Jun N-terminal Kinase/Stress-activated Protein Kinases in the Presence of the Constitutively Active GTP-binding Protein Rac1

This publication has 36 references indexed in Scilit:

• Inhibition of Bax Channel-Forming Activity by Bcl-2
  Science, 1997
• Increased cyclin B1/CDC 2 kinase activity and phosphorylation of Bcl-2 associated with paclitaxel-induced apoptosis in human nasopharyngeal carcinoma cells
  Apoptosis, 1996
• The stress-activated protein kinase pathway mediates cell death following injury induced by cis-platinum, UV irradiation or heat
  Current Biology, 1996
• Requirement for ceramide-initiated SAPK/JNK signalling in stress-induced apoptosis
  Nature, 1996
• New members of the Bcl-2 family and their protein partners
  Current Opinion in Genetics & Development, 1996
• Opposing Effects of ERK and JNK-p38 MAP Kinases on Apoptosis
  Science, 1995
• The small GTP-binding proteins Rac1 and Cdc42regulate the activity of the JNK/SAPK signaling pathway
  Cell, 1995
• Parallel signal processing among mammalian MAPKs
  Trends in Biochemical Sciences, 1995
• Apoptosis in the Pathogenesis and Treatment of Disease
  Science, 1995
• Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4
  Nature, 1970