Receptor Interacting Protein Kinase-3 Determines Cellular Necrotic Response to TNF-α
Top Cited Papers
- 1 June 2009
- Vol. 137 (6) , 1100-1111
- https://doi.org/10.1016/j.cell.2009.05.021
Abstract
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This publication has 35 references indexed in Scilit:
- Identification of a Molecular Signaling Network that Regulates a Cellular Necrotic Cell Death PathwayCell, 2008
- TNF-α Induces Two Distinct Caspase-8 Activation PathwaysCell, 2008
- Autocrine TNFα Signaling Renders Human Cancer Cells Susceptible to Smac-Mimetic-Induced ApoptosisCancer Cell, 2007
- Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell deathNature, 2005
- A Small Molecule Smac Mimic Potentiates TRAIL- and TNFα-Mediated Cell DeathScience, 2004
- Tumor Necrosis Factor-induced Nonapoptotic Cell Death Requires Receptor-interacting Protein-mediated Cellular Reactive Oxygen Species AccumulationJournal of Biological Chemistry, 2004
- The CD95(APO-1/Fas) DISC and beyondCell Death & Differentiation, 2003
- Identification of a Novel Homotypic Interaction Motif Required for the Phosphorylation of Receptor-interacting Protein (RIP) by RIP3Journal of Biological Chemistry, 2002
- Identification of RIP3, a RIP-like kinase that activates apoptosis and NFκBCurrent Biology, 1999
- TNF-Dependent Recruitment of the Protein Kinase RIP to the TNF Receptor-1 Signaling ComplexImmunity, 1996