IKK-i/IKKϵ Controls Constitutive, Cancer Cell-associated NF-κB Activity via Regulation of Ser-536 p65/RelA Phosphorylation
Open Access
- 1 September 2006
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 281 (37) , 26976-26984
- https://doi.org/10.1074/jbc.m603133200
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Tuning up inflammation: How DNA sequence and chromatin organization control the induction of inflammatory genes by NF‐κBFEBS Letters, 2006
- Post-translational modification of RelA(p65) NF-κBBiochemical Society Transactions, 2004
- Signaling to NF-κBGenes & Development, 2004
- One Nucleotide in a κB Site Can Determine Cofactor Specificity for NF-κB DimersCell, 2004
- Interferon response induced by Toll-like receptor signalingInnate Immunity, 2004
- Akt Stimulates the Transactivation Potential of the RelA/p65 Subunit of NF-κB through Utilization of the IκB Kinase and Activation of the Mitogen-activated Protein Kinase p38Journal of Biological Chemistry, 2001
- IKKε Is Part of a Novel PMA-Inducible IκB Kinase ComplexMolecular Cell, 2000
- IKK-i, a novel lipopolysaccharide-inducible kinase that is related to IκB kinasesInternational Immunology, 1999
- Activation of Nuclear Factor-κB-dependent Transcription by Tumor Necrosis Factor-α Is Mediated through Phosphorylation of RelA/p65 on Serine 529Journal of Biological Chemistry, 1998
- Phosphorylation of NF-κB p65 by PKA Stimulates Transcriptional Activity by Promoting a Novel Bivalent Interaction with the Coactivator CBP/p300Molecular Cell, 1998