Evidence That the Pertussis Toxin-sensitive Trimeric GTP-binding Protein Gi2 Is Required for Agonist- and Store-activated Ca2+ Inflow in Hepatocytes

This publication has 20 references indexed in Scilit:

• Chromatographic Resolution of an Intracellular Calcium Influx Factor from Thapsigargin-activated Jurkat Cells
  Published by Elsevier ,1995
• Calcium signaling
  Cell, 1995
• Evidence from studies with hepatocyte suspensions that store-operated Ca2+ inflow requires a pertussis toxin-sensitive trimeric G-protein
  Biochemical Journal, 1994
• A role for a pertussis toxin‐sensitive trimeric G‐protein in store‐operated Ca²⁺inflow in hepatocytes
  FEBS Letters, 1994
• A slowly ADP-ribosylated pertussis-toxin-sensitive GTP-binding regulatory protein is required for vasopressin-stimulated Ca2+ inflow in hepatocytes
  Biochemical Journal, 1994
• Ion Channels and Calcium Signaling in Mast Cells^a
  Annals of the New York Academy of Sciences, 1993
• The signal for capacitative calcium entry
  Cell, 1993
• Depletion of InsP3 stores activates a Ca2+ and K+ current by means of a phosphatase and a diffusible messenger
  Nature, 1993
• Emptying of intracellular Ca2+ stores releases a novel small messenger that stimulates Ca2+ influx
  Nature, 1993
• Inositol phosphates and Ca ²⁺ entry: toward a proliferation or a simplification?
  The FASEB Journal, 1992