Troglitazone, a Peroxisome Proliferator-activated Receptor γ (PPARγ) Ligand, Selectively Induces the Early Growth Response-1 Gene Independently of PPARγ
Open Access
- 1 February 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (8) , 5845-5853
- https://doi.org/10.1074/jbc.m208394200
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- Troglitazone Inhibits Growth of MCF-7 Breast Carcinoma Cells by Targeting G1 Cell Cycle RegulatorsBiochemical and Biophysical Research Communications, 2001
- Induction of Egr-1 expression by the retinoid AHPN in human lung carcinoma cells is dependent on activated ERK1/2Cell Death & Differentiation, 2001
- Combinations of ERK and p38 MAPK Inhibitors Ablate Tumor Necrosis Factor-α (TNF-α) mRNA InductionJournal of Biological Chemistry, 2001
- Effects of Troglitazone on Cellular Differentiation, Insulin Signaling, and Glucose Metabolism in Cultured Human Skeletal Muscle CellsBiochemical and Biophysical Research Communications, 2001
- The Transcription Factor EGR-1 Directly Transactivates the Fibronectin Gene and Enhances Attachment of Human Glioblastoma Cell Line U251Journal of Biological Chemistry, 2000
- Troglitazone Suppresses Cell Growth of Myeloid Leukemia Cell Lines by Induction of p21WAF1/CIP1 Cyclin-Dependent Kinase InhibitorBiochemical and Biophysical Research Communications, 1999
- Peroxisome Proliferator‐activated Receptor γ Induces Growth Arrest and Differentiation Markers of Human Colon Cancer CellsJapanese Journal of Cancer Research, 1999
- PPARγ3 mRNA: a distinct PPARγ mRNA subtype transcribed from an independent promoterFEBS Letters, 1998
- Decreased Egr-1 expression in human, mouse and rat mammary cells and tissues correlates with tumor formationInternational Journal of Cancer, 1997
- Inhibition of Adipogenesis Through MAP Kinase-Mediated Phosphorylation of PPARγScience, 1996