Unc93B1 Restricts Systemic Lethal Inflammation by Orchestrating Toll-like Receptor 7 and 9 Trafficking

This publication has 52 references indexed in Scilit:

• Dendritic Cells in Lupus Are Not Required for Activation of T and B Cells but Promote Their Expansion, Resulting in Tissue Damage
  Immunity, 2010
• A critical role for IL-21 receptor signaling in the pathogenesis of systemic lupus erythematosus in BXSB-Yaamice
  Proceedings of the National Academy of Sciences, 2009
• The costimulatory molecule ICOS regulates the expression of c-Maf and IL-21 in the development of follicular T helper cells and TH-17 cells
  Nature Immunology, 2008
• IRAK-4- and MyD88-Dependent Pathways Are Essential for the Removal of Developing Autoreactive B Cells in Humans
  Immunity, 2008
• UNC93B1 delivers nucleotide-sensing toll-like receptors to endolysosomes
  Nature, 2008
• Control of Toll-like Receptor 7 Expression Is Essential to Restrict Autoimmunity and Dendritic Cell Proliferation
  Immunity, 2007
• Toll-like Receptor 7 and TLR9 Dictate Autoantibody Specificity and Have Opposing Inflammatory and Regulatory Roles in a Murine Model of Lupus
  Published by Elsevier ,2006
• ATlr7translocation accelerates systemic autoimmunity in murine lupus
  Proceedings of the National Academy of Sciences, 2006
• The Unc93b1 mutation 3d disrupts exogenous antigen presentation and signaling via Toll-like receptors 3, 7 and 9
  Nature Immunology, 2006
• Intracellular localization of Toll-like receptor 9 prevents recognition of self DNA but facilitates access to viral DNA
  Nature Immunology, 2005