Open-Channel Block by the Cytoplasmic Tail of Sodium Channel β4 as a Mechanism for Resurgent Sodium Current
- 1 January 2005
- Vol. 45 (2) , 233-244
- https://doi.org/10.1016/j.neuron.2004.12.035
Abstract
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This publication has 45 references indexed in Scilit:
- Heterophilic Interactions of Sodium Channel β1 Subunits with Axonal and Glial Cell Adhesion MoleculesPublished by Elsevier ,2004
- Resurgent Na Currents in Four Classes of Neurons of the CerebellumJournal of Neurophysiology, 2004
- Sodium Channel β1 Subunit-mediated Modulation of Nav1.2 Currents and Cell Surface Density Is Dependent on Interactions with Contactin and AnkyrinPublished by Elsevier ,2004
- Production of Resurgent Current in NaV1.6-Null Purkinje Neurons by Slowing Sodium Channel Inactivation with β-PompilidotoxinJournal of Neuroscience, 2004
- Cloning, Localization, and Functional Expression of Sodium Channel β1A SubunitsPublished by Elsevier ,2000
- Molecular basis of fast inactivation in voltage and Ca 2+ -activated K + channels: A transmembrane β-subunit homologProceedings of the National Academy of Sciences, 1999
- Role of an S4-S5 linker in sodium channel inactivation probed by mutagenesis and a peptide blocker.The Journal of general physiology, 1996
- Internal block of human heart sodium channels by symmetrical tetra-alkylammoniums.The Journal of general physiology, 1994
- Restoration of inactivation and block of open sodium channels by an inactivation gate peptideNeuron, 1994
- Inactivation properties of voltage-gated K+ channels altered by presence of β-subunitNature, 1994