PKC412 overcomes resistance to imatinib in a murine model of FIP1L1-PDGFRα-induced myeloproliferative disease
Open Access
- 1 May 2003
- journal article
- research article
- Published by Elsevier in Cancer Cell
- Vol. 3 (5) , 459-469
- https://doi.org/10.1016/s1535-6108(03)00108-9
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- A Tyrosine Kinase Created by Fusion of thePDGFRAandFIP1L1Genes as a Therapeutic Target of Imatinib in Idiopathic Hypereosinophilic SyndromeNew England Journal of Medicine, 2003
- Novel translocations that disrupt the platelet‐derived growth factor receptor β (PDGFRB) gene in BCR–ABL‐negative chronic myeloproliferative disordersBritish Journal of Haematology, 2003
- Response of idiopathic hypereosinophilic syndrome to treatment with imatinib mesylateLeukemia Research, 2002
- Response to Imatinib Mesylate in Patients with Chronic Myeloproliferative Diseases with Rearrangements of the Platelet-Derived Growth Factor Receptor BetaNew England Journal of Medicine, 2002
- Glivec (STI571, imatinib), a rationally developed, targeted anticancer drugNature Reviews Drug Discovery, 2002
- The t(4;22)(q12;q11) in atypical chronic myeloid leukaemia fuses BCR to PDGFRAHuman Molecular Genetics, 2002
- Protein kinases — the major drug targets of the twenty-first century?Nature Reviews Drug Discovery, 2002
- Induction of Chronic Myelogenous Leukemia in Mice by the P210
bcr/abl
Gene of the Philadelphia ChromosomeScience, 1990
- Transformation of an interleukin 3-dependent hematopoietic cell line by the chronic myelogenous leukemia-specific P210bcr/abl protein.Proceedings of the National Academy of Sciences, 1988
- THE HYPEREOSINOPHILIC SYNDROMEMedicine, 1975