Identification of β-Arrestin2 as a G Protein-Coupled Receptor-Stimulated Regulator of NF-κB Pathways
- 1 May 2004
- journal article
- research article
- Published by Elsevier in Molecular Cell
- Vol. 14 (3) , 303-317
- https://doi.org/10.1016/s1097-2765(04)00216-3
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Modulation of Th1 Activation and Inflammation by the NF-κB Repressor Foxj1Science, 2004
- Myocyte Adrenoceptor Signaling PathwaysScience, 2003
- Pharmacology and Physiology of Human Adrenergic Receptor polymorphismsAnnual Review of Pharmacology and Toxicology, 2003
- β-Arrestin2 Is Critically Involved in CXCR4-mediated Chemotaxis, and This Is Mediated by Its Enhancement of p38 MAPK ActivationJournal of Biological Chemistry, 2002
- β-Arrestins regulate a Ral-GDS–Ral effector pathway that mediates cytoskeletal reorganizationNature Cell Biology, 2002
- Neuroendocrine Regulation of ImmunityAnnual Review of Immunology, 2002
- NF-κB at the crossroads of life and deathNature Immunology, 2002
- A Subclass of Ras Proteins That Regulate the Degradation of IκBScience, 2000
- THE NF-κB AND IκB PROTEINS: New Discoveries and InsightsAnnual Review of Immunology, 1996
- Catecholamine Influences and Sympathetic Neural Modulation of Immune ResponsivenessAnnual Review of Pharmacology and Toxicology, 1995