Autoreceptor presynaptic control of dopamine release from striatum is lost at early stages of manganese poisoning
- 21 April 1995
- journal article
- Published by Elsevier in Life Sciences
- Vol. 56 (22) , 1857-1864
- https://doi.org/10.1016/0024-3205(95)00160-8
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- NMDA receptor mediates dopamine release in the striatum of unanesthetized rats as measured by brain microdialysisBrain Research, 1992
- Dopamine Agonists Suppress Acetylcholine Release from Striatum but Not from Olfactory TubercleAnnals of the New York Academy of Sciences, 1986
- Chemical and structural analysis of the relation between cortical inputs and tyrosine hydroxylase-containing terminals in rat neostriatumBrain Research, 1984
- The Effects of Chronic Manganese Chloride Treatment Expressed as Age-Dependent, Transient Changes in Rat Brain Synaptosomal Uptake of AminesJournal of Neurochemistry, 1982
- Striatal glutamatergic function: Modifications following specific lesionsBrain Research, 1982
- Phenoxybenzamine blocks dopamine autoreceptors irreversibly: Implications for multiple dopamine receptor hypothesesEuropean Journal of Pharmacology, 1981
- Striatal dopamine turnover and l-dopa treatment after short-term exposure of rats to manganeseArchives of Toxicology, 1981
- Interactions Between Manganese and Brain DopamineMedical Clinics of North America, 1976
- EFFECT OF l‐DOPA ON BRAIN CONCENTRATION OF DOPAMINE AND HOMOVANILLIC ACID IN RATS AFTER CHRONIC MANGANESE CHLORIDE ADMINISTRATIONJournal of Neurochemistry, 1974
- Chronic manganese poisoningNeurology, 1967