A Novel Mechanism of Pacemaker Control That Depends on High Levels of cAMP and PKA-Dependent Phosphorylation

This publication has 11 references indexed in Scilit:

• High Basal Protein Kinase A–Dependent Phosphorylation Drives Rhythmic Internal Ca ²⁺ Store Oscillations and Spontaneous Beating of Cardiac Pacemaker Cells
  Circulation Research, 2006
• Ca ²⁺ Sparks and Waves in Canine Purkinje Cells
  Circulation Research, 2005
• Rhythmic Ca²⁺Oscillations Drive Sinoatrial Nodal Cell Pacemaker Function to Make the Heart Tick
  Annals of the New York Academy of Sciences, 2005
• Intracellular Ca²⁺ and pacemaking within the rabbit sinoatrial node: heterogeneity of role and control
  The Journal of Physiology, 2004
• Rhythmic Ryanodine Receptor Ca ²⁺ Releases During Diastolic Depolarization of Sinoatrial Pacemaker Cells Do Not Require Membrane Depolarization
  Circulation Research, 2004
• Diastolic Calcium Release Controls the Beating Rate of Rabbit Sinoatrial Node Cells: Numerical Modeling of the Coupling Process
  Biophysical Journal, 2004
• The Role of Calsequestrin, Triadin, and Junctin in Conferring Cardiac Ryanodine Receptor Responsiveness to Luminal Calcium
  Biophysical Journal, 2004
• Ca ²⁺ Sparks in Rabbit Ventricular Myocytes Evoked by Action Potentials
  Circulation Research, 2003
• Modulation of CICR has no maintained effect on systolic Ca²⁺: simultaneous measurements of sarcoplasmic reticulum and sarcolemmal Ca²⁺ fluxes in rat ventricular myocytes
  The Journal of Physiology, 2000
• The ionic currents underlying pacemaker activity in rabbit sino-atrial node: experimental results and computer simulations
  Proceedings of the Royal Society of London. B. Biological Sciences, 1984