Low hepatic glutathione S-transferase and increased hepatic DNA adduction contribute to increased tumorigenicity of aflatoxin B1 in newborn and partially hepatectomized mice
- 1 March 2004
- journal article
- Published by Elsevier in Toxicology Letters
- Vol. 148 (1-2) , 1-9
- https://doi.org/10.1016/j.toxlet.2003.11.008
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Glutathione- S -transferase activity toward aflatoxin epoxide in livers of mastomys and other rodentsToxicon, 2002
- Mutational properties of the primary aflatoxin B1-DNA adduct.Proceedings of the National Academy of Sciences, 1996
- Induction of Phase I and Phase II Drug-Metabolizing Enzyme mRNA, Protein, and Activity by BHA, Ethoxyquin, and OltiprazToxicology and Applied Pharmacology, 1995
- Mechanisms of Aflatoxin CarcinogenesisAnnual Review of Pharmacology and Toxicology, 1994
- Neonatal Modulation of Hepatic Acid Soluble Sulfhydryls and Xenobiotic Metabolizing Enzymes in Suckling Mice Exposed Translactationally to Butylated HydroxyanisoleDevelopmental Pharmacology and Therapeutics, 1992
- Effect of proliferative state on glutathione S-transferase isoenzyme in cultured rat liver epithelial cellsCarcinogenesis: Integrative Cancer Research, 1991
- The development of hepatocellular tumours following aflatoxin B1 exposure of the partially hepatectomised mouseCarcinogenesis: Integrative Cancer Research, 1984
- Distribution of [14C]‐Labelled Aflatoxin B1 in MiceActa Pharmacologica et Toxicologica, 1978
- Identification of the principal aflatoxin B1-DNA adduct formed in vivo in rat liver.Proceedings of the National Academy of Sciences, 1978
- Biliary Excretion of Aflatoxin in the Rat after a Single DoseNature, 1967