Acute ethanol intoxication regulates f-met-leu-phe-induced chemotaxis and superoxide release by neutrophils and Kupffer cells through modulation of the formyl peptide receptor in the rat
- 1 January 1994
- journal article
- Published by Elsevier in Life Sciences
- Vol. 54 (11) , 721-730
- https://doi.org/10.1016/0024-3205(94)90161-9
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Modulation of F-met-leu-phe Induced Chemotactic Activity and Superoxide Production by Neutrophils during Chronic Ethanol IntoxicationAlcohol, Clinical and Experimental Research, 1992
- Acute Ethanol Intoxication Stimulates Superoxide Anion Production By In Situ Perfused Rat LiverHepatology, 1992
- Introduction: Role of lipid peroxidation and oxidative stress in alcohol toxicityFree Radical Biology & Medicine, 1989
- Biphasic control of polymorphonuclear cell migration by Kupffer cells. Effect of exposure to metabolic products of ethanolLife Sciences, 1988
- Pathophysiology of alcoholic liver diseaseMolecular Aspects of Medicine, 1988
- Alcohol-Induced Hepatotoxicity: A Role for Oxygen Free RadicalsFree Radical Research Communications, 1987
- A chemoattractant receptor on macrophages exists in two affinity states regulated by guanine nucleotides.The Journal of cell biology, 1984
- Increased superoxide anion production by immunologically activated and chemically elicited macrophagesThe Journal of Experimental Medicine, 1978
- Isolation and characterization of Kupffer and endothelial cells from the rat liverExperimental Cell Research, 1977
- THE ATTRACTIONS OF PROTEINS FOR SMALL MOLECULES AND IONSAnnals of the New York Academy of Sciences, 1949