Abstract
The histamine release induced in isolated perfused sensitised guinea-pig lungs by antigen, trypsin, Russell viper venom, and compound 48/80 has been compared. At equi-active dosage for histamine release, these four substances released varying amounts of slow reacting substances. Neither histamine release nor the release of slow reacting substances appeared to be responsible for the changes in cholesterol, glyceride or lipid phosphorus of the lung tissue observed in these experiments.