TRAUMATIC SHOCK. V. A STUDY OF THE EFFECT OF OXYGEN ON HEMORRHAGIC SHOCK

Abstract
The course of events in hemorrhagic shock is in no way altered when [image] venous anoxemia is prevented by administering O2 at high pressure. Tissue anoxia in these circumstances may still exist, presumably because peripheral stagnation results in what amounts to the existence of an arterio-venous shunt. O2 as a therapeutic agent in hemorrhagic shock is therefore of doubtful value. Further evidence is given that the velocity of peripheral blood flow in hemorrhagic shock is reduced.

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