Distinct cardiodynamic and molecular characteristics during early and late stages of sepsis-induced myocardial dysfunction

This publication has 45 references indexed in Scilit:

• CARDIOMYOCYTIC APOPTOSIS FOLLOWING GLOBAL CARDIAC ISCHEMIA AND REPERFUSION CAN BE ATTENUATED BY PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR α BUT NOT γ ACTIVATORS
  Shock, 2006
• Bigendothelin-1 via p38-MAPK-dependent mechanism regulates adult rat ventricular myocyte contractility in sepsis
  Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2005
• Left ventricular mitogen activated protein kinase signaling following polymicrobial sepsis during streptozotocin-induced hyperglycemia
  Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2004
• Metalloendopeptidase Inhibition Regulates Phosphorylation of p38???Mitogen-Activated Protein Kinase and Nitric Oxide Synthase in Heart After Endotoxemia
  Shock, 2003
• Progression From Compensated Hypertrophy to Failure in the Pressure-Overloaded Human Heart
  Circulation, 2003
• Proteasome-mediated Degradation of Smac during Apoptosis: XIAP Promotes Smac Ubiquitination in Vitro
  Journal of Biological Chemistry, 2002
• Interleukin-1β Regulation of Inducible Nitric Oxide Synthase and Cyclooxygenase-2 Involves the p42/44 and p38 MAPK Signaling Pathways in Cardiac Myocytes
  Hypertension, 1999
• Stimulation of the p38 Mitogen-activated Protein Kinase Pathway in Neonatal Rat Ventricular Myocytes by the G Protein–coupled Receptor Agonists, Endothelin-1 and Phenylephrine: A Role in Cardiac Myocyte Hypertrophy?
  The Journal of cell biology, 1998
• Opposing Effects of ERK and JNK-p38 MAP Kinases on Apoptosis
  Science, 1995
• Norepinephrine alone versus norepinephrine plus low-dose dopamine
  Critical Care Medicine, 1985