Golli Protein Negatively Regulates Store Depletion-Induced Calcium Influx in T Cells
- 1 June 2006
- Vol. 24 (6) , 717-727
- https://doi.org/10.1016/j.immuni.2006.04.007
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (NS23022, NS33091)
- National Multiple Sclerosis Society (NS23022, NS33091)
This publication has 39 references indexed in Scilit:
- STIM1 is a Ca2+ sensor that activates CRAC channels and migrates from the Ca2+ store to the plasma membraneNature, 2005
- A severe defect in CRAC Ca2+ channel activation and altered K+ channel gating in T cells from immunodeficient patientsThe Journal of Experimental Medicine, 2005
- STIM Is a Ca2+ Sensor Essential for Ca2+-Store-Depletion-Triggered Ca2+ InfluxCurrent Biology, 2005
- T-cell anergyCurrent Opinion in Immunology, 2004
- Transcriptional Mechanisms Underlying Lymphocyte ToleranceCell, 2002
- MAP Kinases in the Immune ResponseAnnual Review of Immunology, 2002
- Calcium Signaling Mechanisms in T LymphocytesAnnual Review of Immunology, 2001
- PKC-θ is required for TCR-induced NF-κB activation in mature but not immature T lymphocytesNature, 2000
- Defective Thymocyte Maturation in p44 MAP Kinase (Erk 1) Knockout MiceScience, 1999
- Transgenic mice that express a myelin basic protein-specific T cell receptor develop spontaneous autoimmunityCell, 1993