An activating mutation in the transmembrane domain of the granulocyte colony-stimulating factor receptor in patients with acute myeloid leukemia
- 5 September 2002
- journal article
- Published by Springer Nature in Oncogene
- Vol. 21 (39) , 5981-5989
- https://doi.org/10.1038/sj.onc.1205767
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 45 references indexed in Scilit:
- STAT-3 Activation Is Required for Normal G-CSF-Dependent Proliferation and Granulocytic DifferentiationImmunity, 2001
- Down-Regulation of Cell Surface Receptors Is Modulated by Polar Residues within the Transmembrane DomainMolecular Biology of the Cell, 2000
- RTK mutations and human syndromesTrends in Genetics, 2000
- Stat3 as an OncogenePublished by Elsevier ,1999
- An Improved Method for Generating Retroviral Producer Clones for Vectors Lacking a Selectable Marker GeneBlood Cells, Molecules, and Diseases, 1998
- Expression of granulocyte colony‐stimulating factor receptor increases with differentiation in myeloid cells by a newly‐devised quantitative flow‐cytometric assayBritish Journal of Haematology, 1995
- Dimerization of cell surface receptors in signal transductionCell, 1995
- Point mutation in the exoplasmic domain of the erythropoietin receptor resulting in hormone-independent activation and tumorigenicityNature, 1990
- Analysis of RAS gene mutations in acute myeloid leukemia by polymerase chain reaction and oligonucleotide probes.Proceedings of the National Academy of Sciences, 1988
- Multiple independent activations of the neu oncogene by a point mutation altering the transmembrane domain of p185Cell, 1986