Human mitochondrial transcription termination exhibits RNA polymerase independence and biased bipolarity in vitro.
Open Access
- 1 November 1994
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 269 (46) , 29112-29120
- https://doi.org/10.1016/s0021-9258(19)62019-3
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Accumulation of mtDNA with a Mutation at Position 3271 in tRNALeu(UUR) Gene Introduced from a Melas Patient to HeLa Cells Lacking mtDNA Results in Progressive Inhibition of Mitochondrial Respiratory FunctionBiochemical and Biophysical Research Communications, 1993
- The DNA replication fork can pass RNA polymerase without displacing the nascent transcriptNature, 1993
- Limited proteolysis unmasks specific DNA-binding of the murine RNA polymerase I-specific transcription termination factor TTFIJournal of Molecular Biology, 1992
- A new mtDNA mutation associated with mitochondrial myopathy, encephalopathy, lactic acidosis and stroke-like episodes (MELAS)Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1991
- Maternally inherited myopathy and cardiomyopathy: association with mutation in mitochondrial DNA tRNALeu(UUR)The Lancet, 1991
- Impairment of mitochondrial transcription termination by a point mutation associated with the MELAS subgroup of mitochondrial encephalomyopathiesNature, 1991
- Specific interaction of the murine transcription termination factor TTF I with class-I RNA polymerasesNature, 1990
- Analysis of the signals for transcription termination by purified RNA polymerase IIBiochemistry, 1990
- Termination of transcription in human mitochondria: Identification and purification of a DNA binding protein factor that promotes terminationCell, 1989
- Transcription of mouse rDNA terminates downstream of the 3′ end of 28S RNA and involves interaction of factors with repeated sequences in the 3′ spacerCell, 1985