Biofilm Formation Induces C3a Release and ProtectsStaphylococcus epidermidisfrom IgG and Complement Deposition and from Neutrophil‐Dependent Killing
Open Access
- 1 April 2008
- journal article
- Published by Oxford University Press (OUP) in The Journal of Infectious Diseases
- Vol. 197 (7) , 1028-1035
- https://doi.org/10.1086/528992
Abstract
BackgroundBiofilm formation is considered to be an important virulence factor of the opportunistic pathogen Staphylococcus epidermidis. We hypothesizKeywords
This publication has 38 references indexed in Scilit:
- Comparative Antibody-Mediated Phagocytosis ofStaphylococcus epidermidisCells Grown in a Biofilm or in the Planktonic StateInfection and Immunity, 2006
- Extracellular Carbohydrate-Containing Polymers of a Model Biofilm-Producing Strain, Staphylococcus epidermidis RP62AInfection and Immunity, 2005
- A Crucial Role for Exopolysaccharide Modification in Bacterial Biofilm Formation, Immune Evasion, and VirulenceJournal of Biological Chemistry, 2004
- Polysaccharide intercellular adhesin (PIA) protects Staphylococcus epidermidis against major components of the human innate immune systemCellular Microbiology, 2004
- Pathogenesis of infections due to coagulasenegative staphylococciThe Lancet Infectious Diseases, 2002
- Staphylococcus and biofilmsMolecular Microbiology, 2002
- Staphylococcus epidermidis biofilms: importance and implicationsJournal of Medical Microbiology, 2001
- Molecular basis of intercellular adhesion in the biofilm‐forming Staphylococcus epidermidisMolecular Microbiology, 1996
- The intercellular adhesin involved in biofilm accumulation of Staphylococcus epidermidis is a linear beta-1,6-linked glucosaminoglycan: purification and structural analysisJournal of Bacteriology, 1996
- The effects of extracellular slime fromStaphylococcus epidermidison phagocytic ingestion and killingFEMS Immunology & Medical Microbiology, 1994