Enhanced Release of Prostaglandins Contributes to Flow-Induced Arteriolar Dilation in eNOS Knockout Mice
- 6 August 1999
- journal article
- other
- Published by Wolters Kluwer Health in Circulation Research
- Vol. 85 (3) , 288-293
- https://doi.org/10.1161/01.res.85.3.288
Abstract
—Nitric oxide and prostaglandins were shown to contribute to the endothelial mediation of flow-induced dilation of skeletal muscle arterioles of rats. Thus, we hypothesized that flow-induce...Keywords
This publication has 21 references indexed in Scilit:
- Acetylcholine‐induced relaxation in blood vessels from endothelial nitric oxide synthase knockout miceBritish Journal of Pharmacology, 1999
- Hypotension and reduced nitric oxide-elicited vasorelaxation in transgenic mice overexpressing endothelial nitric oxide synthase.Journal of Clinical Investigation, 1998
- Adaptation of Flow-Induced Dilation of Arterioles to Daily ExerciseMicrovascular Research, 1998
- Chronic inhibition of NO synthase enhances the production of prostacyclin in coronary arteries through upregulation of the cyclooxygenase type 1 isoformFundamental & Clinical Pharmacology, 1997
- Alteration of flow‐induced dilatation in mesenteric resistance arteries of L‐NAME treated rats and its partial association with induction of cyclo‐oxygenase‐2British Journal of Pharmacology, 1997
- Hypertension in mice lacking the gene for endothelial nitric oxide synthaseNature, 1995
- Flow‐Dependent Dilation and Myogenic Constriction Interact to Establish the Resistance of Skeletal Muscle ArteriolesMicrocirculation, 1995
- Pressure-flow curves reflect arteriolar responses in perfused rat hindquarters.Hypertension, 1994
- Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factorNature, 1987
- The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholineNature, 1980