Transient ischemia causes a reduction of Mg2+ blockade of NMDA receptors
- 23 May 1994
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 173 (1-2) , 75-78
- https://doi.org/10.1016/0304-3940(94)90153-8
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- A selective LTP of NMDA receptor-mediated currents induced by anoxia in CA1 hippocampal neuronsJournal of Neurophysiology, 1993
- Protein kinase C reduces Mg2+ block of NMDA-receptor channels as a mechanism of modulationNature, 1992
- Blockade of intracellular actions of calcium may protect against ischaemic damage to the gerbil brainBritish Journal of Pharmacology, 1991
- The N-methyl-D-aspartate antagonist, MK-801, fails to protect against neuronal damage caused by transient, severe forebrain ischemia in adult ratsJournal of Neuroscience, 1991
- Calpain inhibitors improve the recovery of synaptic transmission from hypoxia in hippocampal slicesBrain Research, 1990
- Neuronal damage and calcium accumulation following repeated brief cerebral ischemia in the gerbilBrain Research, 1990
- Hypothermia but not the N-methyl-D-aspartate antagonist, MK-801, attenuates neuronal damage in gerbils subjected to transient global ischemiaJournal of Neuroscience, 1990
- Blockade of excitatory amino acid receptors protects anoxic hippocampal slicesNeuroscience, 1987
- The reversal potential of excitatory amino acid action on granule cells of the rat dentate gyrus.The Journal of Physiology, 1984
- Excitatory amino acids in synaptic transmission in the Schaffer collateral‐commissural pathway of the rat hippocampus.The Journal of Physiology, 1983