TATA-binding protein in neurodegenerative disease
- 1 January 2005
- journal article
- review article
- Published by Elsevier in Neuroscience
- Vol. 133 (4) , 863-872
- https://doi.org/10.1016/j.neuroscience.2005.03.024
Abstract
No abstract availableKeywords
This publication has 87 references indexed in Scilit:
- Behavioral Disorder, Dementia, Ataxia, and Rigidity in a Large Family With TATA Box-Binding Protein MutationArchives of Neurology, 2004
- Prefibrillar Amyloid Protein Aggregates Share Common Features of CytotoxicityJournal of Biological Chemistry, 2004
- Trinucleotide repeat expansion in SCA17/TBP in white patients with Huntington's disease-like phenotypeJournal of Medical Genetics, 2004
- Huntingtin aggregation and toxicity in Huntington's diseaseThe Lancet, 2003
- Results of a high-resolution genome screen of 437 Alzheimer's Disease familiesHuman Molecular Genetics, 2003
- Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseasesNature, 2002
- Sequestering CBP: the problem with expanded polyglutamine repeatsClinical Genetics, 2001
- Transgenic mice with an expanded CAG repeat controlled by the human AR promoter show polyglutamine nuclear inclusions and neuronal dysfunction without neuronal cell deathHuman Molecular Genetics, 2001
- Expression of expanded repeat androgen receptor produces neurologic disease in transgenic mice.Human Molecular Genetics, 2001
- Five intermediate complexes in transcription initiation by RNA polymerase IICell, 1989