Abstract
Portals of entry via the skin, fins, buccal cavity and digestive tract have been demonstrated for the myxozoan that causes salmonid whirling disease. Experimentally, young rainbow trout Salmo gairdneri were exposed to triactinomyxon spores. Within 10 min after initial exposure, intracellular aggregates of triactinomyxon sporoplasms appeared in the epithelia of exposed fish. During several hours following penetration, the sporoplasms moved or were transported from the external epithelial layers into deeper strata. Unexposed control trout were free of the organism. The aggregates of intracellular triactinomyxon sporoplasms were identified serologically as being Myxobolus cerebralis.
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