AP-1 activation and altered AP-1 composition in association with increased phosphorylation and expression of specific Jun and Fos family proteins induced by vinblastine in KB-3 cells11Abbreviations: JNK, c-Jun NH2-terminal kinase; AP-1, activating protein 1; TPA, 12-O-tetradecanoylphorbol-13-acetate; TRE, TPA response element; DTT, dithiothreitol; PMSF, phenylmethylsulfonyl fluoride; EMSA, electrophoretic mobility shift assay; and TK, thymidine kinase
- 21 August 2001
- journal article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 62 (5) , 581-591
- https://doi.org/10.1016/s0006-2952(01)00694-3
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Requirement of JNK for Stress- Induced Activation of the Cytochrome c-Mediated Death PathwayScience, 2000
- Molecular effects of paclitaxel: Myths and reality (a critical review)International Journal of Cancer, 1999
- The effect of antimicrotubule agents on signal transduction pathways of apoptosis: a reviewCancer Chemotherapy and Pharmacology, 1999
- Dysregulation of Apoptosis in CancerJournal of Clinical Oncology, 1999
- Mitogen-Activated Protein Kinase: Conservation of a Three-Kinase Module From Yeast to HumanPhysiological Reviews, 1999
- Characterization of the p53-Dependent Postmitotic Checkpoint following Spindle DisruptionMolecular and Cellular Biology, 1998
- An Information-Intensive Approach to the Molecular Pharmacology of CancerScience, 1997
- Role of the Stress-activated/c-Jun NH2-terminal Protein Kinase Pathway in the Cellular Response to Adriamycin and Other Chemotherapeutic DrugsPublished by Elsevier ,1996
- Opposing Effects of ERK and JNK-p38 MAP Kinases on ApoptosisScience, 1995
- Inactivation of Bcl-2 by phosphorylation.Proceedings of the National Academy of Sciences, 1995