Hypomagnesaemic hypocalcaemia with hypokalaemia caused by treatment with high dose gentamicin.
- 1 October 1978
- journal article
- research article
- Published by BMJ in Archives of Disease in Childhood
- Vol. 53 (10) , 817-820
- https://doi.org/10.1136/adc.53.10.817
Abstract
A 12-year-old boy developed renal wasting of magnesium, calcium, and potassium, with secondary hypomagnesaemia, hypocalcaemia, and hypokalaaemia (without hyperaldosteronism) after treatment with 14 400 mg gentamicin over 4 months. Gentamicin should not be given for prolonged courses if less toxic antibiotics are suitable. If it used, plasma magnesium, calcium, and potassium levels should be monitored during and after treatment.This publication has 8 references indexed in Scilit:
- GENTAMICIN PERSISTENCE IN THE BODYThe Lancet, 1977
- FUNCTIONAL HYPOPARATHYROIDISM AND PARATHYROID HORMONE END‐ORGAN RESISTANCE IN HUMAN MAGNESIUM DEFICIENCYClinical Endocrinology, 1976
- Experimental aminoglycoside nephrotoxicity.1975
- Hypomagnesemic Hypocalcemia Secondary to Renal Magnesium WastingAnnals of Internal Medicine, 1975
- PROLONGED EXCRETION OF GENTAMICIN IN A PATIENT WITH UNIMPAIRED RENAL FUNCTIONThe Lancet, 1975
- DRUG-INDUCED SECONDARY HYPERALDOSTERONISM IN PATIENTS WITH PULMONARY TUBERCULOSIS1QJM: An International Journal of Medicine, 1970
- Drug-induced secondary hyperaldosteronism in patients with pulmonary tuberculosis.1970
- EXPERIMENTAL HUMAN MAGNESIUM DEPLETIONMedicine, 1969