Differential Involvement of Caspases in Hydroquinone-Induced Apoptosis in Human Leukemic HL-60 and Jurkat Cells
- 1 September 2001
- journal article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 175 (2) , 95-103
- https://doi.org/10.1006/taap.2001.9221
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Oxidative stress as a mediator of apoptosisPublished by Elsevier ,2002
- THE ROLE OF METABOLISM AND SPECIFIC METABOLITES IN BENZENE-INDUCED TOXICITY: EVIDENCE AND ISSUESJournal of Toxicology and Environmental Health, Part A, 2000
- Inhibition of Caspase-3 by S-Nitrosation and Oxidation Caused by Nitric OxideBiochemical and Biophysical Research Communications, 1997
- Processing/Activation of At Least Four Interleukin-1β Converting Enzyme–like Proteases Occurs during the Execution Phase of Apoptosis in Human Monocytic Tumor CellsThe Journal of cell biology, 1997
- Metabolic basis of benzene toxicityEuropean Journal of Haematology, 1996
- Interaction of p-benzoquinone and p-biphenoquinone with microtubule proteins in vitroChemico-Biological Interactions, 1996
- Inhibition of Human T Lymphoblast Proliferation by HydroquinoneToxicology and Applied Pharmacology, 1996
- Benzene metabolites enhance reactive oxygen species generation in HL60 human leukemia cellsHuman & Experimental Toxicology, 1996
- Peroxidase-dependent metabolism of benzene's phenolic metabolites and its potential role in benzene toxicity and carcinogenicity.Environmental Health Perspectives, 1989
- Benzene disposition in the rat after exposure by inhalationToxicology and Applied Pharmacology, 1979