Possible Causes of Alzheimer's Disease: Amyloid Fragments, Free Radicals, and Calcium Homeostasis
- 1 September 1998
- journal article
- review article
- Published by Elsevier in Neurobiology of Disease
- Vol. 5 (3) , 129-141
- https://doi.org/10.1006/nbdi.1998.0193
Abstract
No abstract availableKeywords
This publication has 127 references indexed in Scilit:
- Decay characteristics of HIV-1-infected compartments during combination therapyNature, 1997
- Changes of p53 in the Brains of Patients with Alzheimer's DiseaseBiochemical and Biophysical Research Communications, 1997
- Amyloid β Toxicity Consists of a Ca2+‐Independent Early Phase and a Ca2+‐Dependent Late PhaseJournal of Neurochemistry, 1996
- Actions of Neurotoxic β‐Amyloid on Calcium Homeostasis and Viability of PC12 Cells Are Blocked by Antioxidants but Not by Calcium Channel AntagonistsJournal of Neurochemistry, 1996
- β-Amyloid selectively augments NMDA receptor-mediated synaptic transmission in rat hippocampusNeuroReport, 1995
- Effects of aging and of Alzheimer's disease on verbal memoryJournal of Clinical and Experimental Neuropsychology, 1995
- Opposing regulation of amyloid precursor protein by ionotropic and metabotropic glutamate receptorsNeuroReport, 1995
- β-Amyloid 25–35 activates nitric oxide synthase in a neuronal cloneNeuroReport, 1993
- Neuropathological stageing of Alzheimer-related changesActa Neuropathologica, 1991
- Intraventricular infusions of antibodies to amyloid-β-protein precursor impair the acquisition of a passive avoidance response in the ratNeuroscience Letters, 1990