Bmi-1 cooperates with human papillomavirus type 16 E6 to immortalize normal human oral keratinocytes
- 28 October 2006
- journal article
- Published by Elsevier in Experimental Cell Research
- Vol. 313 (3) , 462-472
- https://doi.org/10.1016/j.yexcr.2006.10.025
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Alteration of cell-cycle regulatory proteins in human oral epithelial cells immortalized by HPV16 E6 and E7International Journal of Oral & Maxillofacial Surgery, 2006
- Bmi-1 Is a Novel Molecular Marker of Nasopharyngeal Carcinoma Progression and Immortalizes Primary Human Nasopharyngeal Epithelial CellsCancer Research, 2006
- Genome-wide mapping of Polycomb target genes unravels their roles in cell fate transitionsGenes & Development, 2006
- Genomic and Proteomic Expression Patterns in HPV-16 E6 Gene Transfected Stable Human Carcinoma Cell LinesDNA and Cell Biology, 2004
- The Wilms Tumor Suppressor-1 Target Gene Podocalyxin Is Transcriptionally Repressed by p53Published by Elsevier ,2004
- Degradation of p53, Not Telomerase Activation, by E6 Is Required for Bypass of Crisis and Immortalization by Human Papillomavirus Type 16 E6/E7Journal of Virology, 2004
- CCR5 Expression Influences the Progression of Human Breast Cancer in a p53-dependent MannerThe Journal of Experimental Medicine, 2003
- Identification of molecular subtypes of glioblastoma by gene expression profilingOncogene, 2003
- Papillomaviruses and cancer: from basic studies to clinical applicationNature Reviews Cancer, 2002
- Specific Association of Human Telomerase Activity with Immortal Cells and CancerScience, 1994