A Mechanism for the Suppression of Ongoing IgE Synthesis

Abstract
IgE synthesis from the human plasmacytoma U266/AF-10 was suppressed by addition of IgE immune complexes (IgE-IC). This suppression was isotype-specific as synthesis from other B cell lines was unaffected. Using IgE-IC constructed with a monoclonal antibody that recognizes PS protein-IgE and not ND IgE (the IgE protein made by U266/AF-10), we have shown that this suppression was mediated through the cross-linking of the Fc epsilon receptor.

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