Abstract
In 1979, Henry Balfour termed cytomegalovirus (CMV) the “troll of transplantation” for the way CMV unwittingly sneaks up on a successful transplant recipient and either precipitates one of its many indirect effects or directly harms the patient [1]. Although we have decreased, rather dramatically, the clinical impact of CMV on organ transplantation [2, 3], with dramatic improvement in patient and graft survival, we know that CMV infection and disease still occur, as is nicely illustrated in the article by San Juan et al. [4] in this issue of the Clinical Infectious Diseases.

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