Abstract
A comparison of 4 host strains for assay of dry T1 phage plaque survival indicates that about 25% of fast electron direct action damage expressed in uvrB-hosts is repaired in uvrB+ hosts. The repair is not associated with a lex or exr gene. In relative amounts, the uvr-repairable damage corresponds with that reported for the T1 phage irradiated by X-rays in heavily protected anoxic suspension and assayed for plaque survival on uvr+ and uvr- host strains.