Thyroid-stimulating Hormone/cAMP and Glycogen Synthase Kinase 3β Elicit Opposing Effects on Rap1GAP Stability
Open Access
- 1 February 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (7) , 5501-5507
- https://doi.org/10.1074/jbc.m305824200
Abstract
No abstract availableKeywords
This publication has 62 references indexed in Scilit:
- The specificities of protein kinase inhibitors: an updateBiochemical Journal, 2003
- Identification of a Wnt/ -Catenin Signaling Pathway in Human Thyroid CellsEndocrinology, 2001
- Regulation of Thyroid Cell Proliferation by TSH and Other Factors: A Critical Evaluation of in Vitro ModelsEndocrine Reviews, 2001
- Rap1 signalling: adhering to new modelsNature Reviews Molecular Cell Biology, 2001
- Wnt/β-Catenin Signaling Induces the Expression and Activity of βTrCP Ubiquitin Ligase ReceptorMolecular Cell, 2000
- Human SPA-1 Gene Product Selectively Expressed in Lymphoid Tissues Is a Specific GTPase-activating Protein for Rap1 and Rap2Journal of Biological Chemistry, 1997
- Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase BNature, 1995
- Identification of Tuberin, the Tuberous Sclerosis-2 Product. TUBERIN POSSESSES SPECIFIC Rap1GAP ACTIVITYJournal of Biological Chemistry, 1995
- Phosphorylation of Rap1GAP during the Cell CycleBiochemical and Biophysical Research Communications, 1994
- Molecular cloning of a GTPase activating protein specific for the Krev-1 protein p21rap1Cell, 1991