Plasma levels of nitric oxide and related vasoactive factors following long-term treatment with angiotensin-converting enzyme inhibitor in patients with essential hypertension
- 1 October 1999
- journal article
- research article
- Published by Elsevier in Metabolism
- Vol. 48 (10) , 1256-1259
- https://doi.org/10.1016/s0026-0495(99)90264-0
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 18 references indexed in Scilit:
- Chronic kinin receptor blockade attenuates the antihypertensive effect of ramipril.Hypertension, 1992
- Ramiprilat enhances endothelial autacoid formation by inhibiting breakdown of endothelium-derived bradykinin.Hypertension, 1991
- Synthesis and release of acetylcholine by cultured bovine arterial endothelial cellsNeuroscience Letters, 1990
- Kinin- and Non-Kinin-Mediated Interactions of Converting Enzyme Inhibitors with Vasoactive HormonesJournal of Cardiovascular Pharmacology, 1990
- Hemodynamic Effects of a Kinin AntagonistJournal of Cardiovascular Pharmacology, 1990
- Role of Kallikrein-Kinin System in the Hypotensive Mechanisms of Converting Enzyme Inhibitors in Essential HypertensionJournal of Cardiovascular Pharmacology, 1989
- Influence of Endogenous Bradykinin on Acute Blood Pressure Response to Vasopressors in Normotensive Rats Assessed with a Bradykinin AntagonistJournal of Cardiovascular Pharmacology, 1988
- Converting enzyme inhibitor ramipril stimulates prostacyclin synthesis by isolated rat aorta: Evidence for a kinin-dependent mechanismJournal of Molecular Medicine, 1986
- Renin- and non-renin–mediated antihypertensive actions of converting enzyme inhibitorsKidney International, 1984
- Converting enzyme inhibition in essential hypertension: the hypotensive response does not reflect only reduced angiotensin II formation.Hypertension, 1979