Effect of valproic acid on 5−hydroxytryptamine turnover in mouse brain

Abstract
The effect of the antiepileptic drug valproic acid (di-n-propylacetic acid, 200 mgkg−1) on brain 5−hydroxytryptamine (5−HT) synthesis during monoamine oxidase inhibition by pargyline hydrochloride (120 mg kg −1) was studied in mice. Valproic acid increased 5−HT synthesis and elevated 5−hydroxyindoleacetic acid level in brain indicating that turnover of 5−HT is increased. The possible significance of this effect in relation to anticonvulsant action of valproic acid is discussed.