Glucuronidation and isomerization of all-trans- and 13-CIS-retinoic acid by liver microsomes of phenobarbital- or 3-methylcholanthrene-treated rats
- 9 February 1994
- journal article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 47 (3) , 485-492
- https://doi.org/10.1016/0006-2952(94)90179-1
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Retinoic Acid: Morphogen or More Mysteries?Nutrition Reviews, 2009
- The high sensitivity of the rabbit to the teratogenic effects of 13-cis-retinoic acid (isotretinoin) is a consequence of prolonged exposure of the embryo to 13-cis-retinoic acid and 13-cis-4-oxo-retinoic acid, and not of isomerization to all-trans-retinoic acidArchives of Toxicology, 1994
- Retinoids in cancer therapy.Journal of Clinical Oncology, 1992
- Glucuronidation and its Role in Regulation of Biological Activity of DrugsAnnual Review of Pharmacology and Toxicology, 1992
- Effects of Retinoid Glucuronides on Mammary Gland Development in Organ CultureOncology, 1991
- Isotretinoin (13‐cis‐retinoic acid) metabolism, cis‐trans isomerization, glucuronidation, and transfer to the mouse embryo: Consequences for teratogenicityTeratogenesis, Carcinogenesis, and Mutagenesis, 1991
- Characterization of oxidized and glucuronidated metabolites of retinol in monkey plasma by thermospray liquid chromatography/mass spectrometryJournal of Mass Spectrometry, 1990
- Vitamin A congenersTeratology, 1986
- Retinoyl β-glucuronide: an endogenous compound of human bloodThe American Journal of Clinical Nutrition, 1986
- Prolonged Remissions of Cystic and Conglobate Acne with 13-cis-Retinoic AcidNew England Journal of Medicine, 1979