Activation of Stress-activated Protein Kinase/c-Jun NH2-terminal Kinase and p38 Kinase in Calphostin C-induced Apoptosis Requires Caspase-3-like Proteases but Is Dispensable for Cell Death
Open Access
- 1 February 1999
- journal article
- Published by Elsevier
- Vol. 274 (9) , 5310-5317
- https://doi.org/10.1074/jbc.274.9.5310
Abstract
No abstract availableKeywords
This publication has 60 references indexed in Scilit:
- SB 203580 is a specific inhibitor of a MAP kinase homologue which is stimulated by cellular stresses and interleukin‐1Published by Wiley ,2000
- Abrogation of c-Raf expression induces apoptosis in tumor cellsOncogene, 1998
- Anti-apoptotic oncogenes prevent caspase-dependent and independent commitment for cell deathCell Death & Differentiation, 1998
- Commitment to cell death measured by loss of clonogenicity is separable from the appearance of apoptotic markersCell Death & Differentiation, 1998
- The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell deathOncogene, 1997
- Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell DeathCell, 1996
- Three paths to stress reliefNature, 1996
- Fibroblast Growth Factor-2 Suppression of Tumor Necrosis Factor α-Mediated Apoptosis Requires Ras and the Activation of Mitogen-activated Protein KinaseJournal of Biological Chemistry, 1996
- CPP32/Apopain Is a Key Interleukin 1β Converting Enzyme-like Protease Involved in Fas-mediated ApoptosisJournal of Biological Chemistry, 1996
- Introduction of soluble protein into the class I pathway of antigen processing and presentationCell, 1988