Cross-talk between 1,25-Dihydroxyvitamin D3 and Transforming Growth Factor-β Signaling Requires Binding of VDR and Smad3 Proteins to Their Cognate DNA Recognition Elements
Open Access
- 1 May 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (19) , 15741-15746
- https://doi.org/10.1074/jbc.m011033200
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Structural and Functional Characterization of the Transforming Growth Factor-β-induced Smad3/c-Jun Transcriptional CooperativityPublished by Elsevier ,2000
- Regulation of Smad ActivityCell, 2000
- Positive and Negative Modulation of Vitamin D Receptor Function by Transforming Growth Factor-β Signaling through Smad ProteinsJournal of Biological Chemistry, 1999
- Convergence of Transforming Growth Factor-β and Vitamin D Signaling Pathways on SMAD Transcriptional CoactivatorsScience, 1999
- Transcriptional Control of Osteoblast DifferentiationThe Endocrinologist, 1999
- Molecular mechanism of vitamin D receptor actionInflammation Research, 1998
- TGF-β signalling through the Smad pathwayTrends in Cell Biology, 1997
- The Missing BoneCell, 1997
- Studies on the Mechanism of Glucocorticoid-Mediated Repression from a Negative Glucocorticoid Response Element from the Bovine Prolactin GeneDNA and Cell Biology, 1997
- 1,25-Dihydroxyvitamin D-Responsive Element and Glucocorticoid Repression in the Osteocalcin GeneScience, 1989