Deficits in NaCl ingestion after damage to the central nucleus of the amygdala in the rat
- 1 January 1992
- journal article
- Published by Elsevier in Brain Research Bulletin
- Vol. 28 (1) , 89-98
- https://doi.org/10.1016/0361-9230(92)90234-o
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Salt appetite and lesions of the ventral part of the ventral median preoptic nucleus.Behavioral Neuroscience, 1990
- The medial amygdala is part of a mineralocorticoid-sensitive circuit controlling NaCl intake in the ratBehavioural Brain Research, 1989
- Interactive responses to stimulation of the amygdaloid central nucleus and baroreceptor afferent activation in the rabbitJournal of the Autonomic Nervous System, 1989
- Organization of Angiotensin II Immunoreactive Cells and Fibers in the Rat Central Nervous SystemNeuroendocrinology, 1985
- Aphagia, adipsia, and sensory-motor deficits produced by amygdala lesions: A function of extra-amygdaloid damage☆Physiology & Behavior, 1977
- Taste pathways to hypothalamus and amygdalaJournal of Comparative Neurology, 1976
- Interaction between hypothalamus, amygdala and septal area in the control of sodium chloride intakePhysiology & Behavior, 1972
- Amygdaloid modulation of sodium chloride and water intake in the rat.Journal of Comparative and Physiological Psychology, 1971
- Role of amygdaloid complex in sodium chloride and water intake in the ratPhysiology & Behavior, 1968
- Disappearence of preferences and aversions for sapid solutions in rats ingesting untasted fluids.Journal of Comparative and Physiological Psychology, 1968