α1-Antichymotrypsin and IL-1β are not increased in CSF or serum in Alzheimer's disease
- 30 June 1994
- journal article
- Published by Elsevier in Neurobiology of Aging
- Vol. 15 (3) , 313-317
- https://doi.org/10.1016/0197-4580(94)90026-4
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- ?1-Antichymotrypsin Binding to Alzheimer A? Peptides Is Sequence Specific and Induces Fibril Disaggregation In VitroJournal of Neurochemistry, 1993
- Localization of TNFα and IL-1a immunoreactivities in striatal neurons after surgical injury to the hippocampusNeuron, 1993
- Cerebral amyloid plaques in Alzheimer’s disease but not in scrapie-affected mice are closely associated with a local inflammatory processVirchows Archiv B Cell Pathology Including Molecular Pathology, 1991
- Interleukin‐6 and α‐2‐macroglobulin indicate an acute‐phase state in Alzheimer's disease corticesFEBS Letters, 1991
- Is amyloidogenesis during Alzheimer's disease due to an IL-1-/IL-6-mediated ‘acute phase response’ in the brain?Immunology Today, 1991
- Occurrence of HLA‐DR Reactive Microglia in Alzheimer's DiseaseAnnals of the New York Academy of Sciences, 1988
- The relevance of the Lewy body to the pathogenesis of idiopathic Parkinson's disease.Journal of Neurology, Neurosurgery & Psychiatry, 1988
- Clinical diagnosis of Alzheimer's diseaseNeurology, 1984
- Principles of albumin and IgG analyses in neurological disorders. I. Establishment of reference valuesScandinavian Journal of Clinical and Laboratory Investigation, 1977
- “Mini-mental state”Journal of Psychiatric Research, 1975