Down-Regulation of Mcl-1 by Inhibition of the PI3-K/Akt Pathway Is Required for Cell Shrinkage-Dependent Cell Death
- 1 February 2002
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 290 (4) , 1275-1281
- https://doi.org/10.1006/bbrc.2002.6329
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Receptor‐mediated control of regulatory volume decrease (RVD) and apoptotic volume decrease (AVD)The Journal of Physiology, 2001
- Normotonic cell shrinkage because of disordered volume regulation is an early prerequisite to apoptosisProceedings of the National Academy of Sciences, 2000
- POSSIBLE INVOLVEMENT OF A CHLORIDE—BICARBONATE EXCHANGER IN APOPTOSIS OF ENDOTHELIAL CELLS AND CARDIOMYOCYTESCell Biology International, 1999
- Activated Phosphatidylinositol 3-Kinase and Akt Kinase Promote Survival of Superior Cervical NeuronsThe Journal of cell biology, 1997
- Caspases: the executioners of apoptosisBiochemical Journal, 1997
- Matrix adhesion and Ras transformation both activate a phosphoinositide 3-OH kinase and protein kinase B/Akt cellular survival pathwayThe EMBO Journal, 1997
- Suppression of c-Myc-induced apoptosis by Ras signalling through PI(3)K and PKBNature, 1997
- Regulation of Neuronal Survival by the Serine-Threonine Protein Kinase AktScience, 1997
- Adrenergic Receptors Activate Phosphatidylinositol 3-Kinase in Human Vascular Smooth Muscle CellsPublished by Elsevier ,1996
- 1-Phosphatidylinositol 3-kinase activity is required for insulin-stimulated glucose transport but not for RAS activation in CHO cells.Proceedings of the National Academy of Sciences, 1994